Pathophysiology Practice Test (17)

Pathophysiology Practice Test (17)

<<<<<<<<<<< Liver Insufficiency>>>>>>>>>>> ................. . Multiple Choice. ......................

published on June 21, 2012
1/15

plasma amino acid imbalance may lead to?

increased production of phenyl-ethanolamine
increased of 5- HT
increased production octopamine
decreased production of dopamine
2/15

hyperammonemia in patient with cirrhosis related to?

inadequate digestion of protein and excess
reproduction of intestinal bacteria
disorder of kidney
alkalosis
increased ammonia formation of the mucles
3/15

the patient with hepatic encephalopathy may have?

Hint: 2 choices
increased AAA ( aromatic amino acid) in plasma
decreased GABA in plasma
decreased BCAA ( branched chain amino acid) in
plasma
decreased phenylalanine in plasma
4/15

imbalance of plasma amino acid may lead to?

Hint: 2 choices
less AAA enter the brain
more BCAA enter the brain
decreased formation of dopamine
increased formation of false neurotransmitter
5/15

the pathogenesis of hepatic ascites is?

portal hypertension
decreased plasma colloidal osmotic pressure
decreased GFR
excessive aldosterone
6/15

the pathogenesis of hepatic encephalopathy result from hyperammonemia are?

Hint: 3 choices
increased consume of ATP
decrease production of ATP
increased formation of false neurotransmitter
increased formation of inhibited neurotransmitter
7/15

what may present in patients with hepatic insufficiency?

encephalopathy
hypoproteinemia
hyperinsulinemia
renal failure
8/15

the pathogenesis of disorder of energy metabolic in brain result from hyperammonemia are?

Hint: 3 choices
increased consume of α - ketoglutaric acid
increased consume of NADH
increased consume of ATP
increased of acetyle-CoA
9/15

patients with hepatic cirrhosis may have?

Hint: 3 choices
increased ADH in the body
increased ALD in the body
increased phagocytic role of kupffer
intestinal endotoxemia may occur cells
10/15

hepatic encephalopathy lead by false neurotransmitter is because?

Hint: 2 choices
FNT may compete with true neurotransmitter
disturb production of energy
FNT accumulated in the synapses of the reticular
structure
increase GABA
11/15

the proper treatment of hepatic encephalopathy is?

L- dopa
reduce oral intake of protein
sterilizing the bowel with antibiotics such as neomycin
acidification of the gut lumen by giving lactulose
12/15

the pathogenesis of hepatic encephalopathy by false neurotransmitter is?

Hint: 2 choices
easily enter blood-brain barrier
there is specific receptor of FNT in membraine of
neuron
FNT may uptake and store and release by neuron
FNT play weak physiological effects
13/15

precipitating causes of hepatic encephalopathy is?

gastrointestinal hemorrhage
alkalosis due to diuretic
constipation
infection
14/15

false neurotransmitter refers to?

Hint: 2 choices
phenyl-ethanolamine
phenyl-ethylamine
octopamine
tyromine
15/15

precipitating factors of hepatic encephalopathy may be?

prerenal azotemia
sedative and anesthetic agents
drink excessive alcohol
operation